Tay-Sachs Disease BINGO Cards - Print Free or Customize
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About: Dive into the intricate world of Tay-Sachs disease and related metabolic disorders with this bingo card designed for genetics students, medical professionals, or rare disease awareness events. The terms featured capture key genetic concepts, disease mechanisms, and diagnostic markers, making it perfect for anyone looking to learn or teach about lysosomal storage diseases in an engaging way.
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How to play Tay-Sachs Disease BINGO Cards?
Printed Players: Print PDF bingo cards and manually scratch the cards.
Printed Caller: Print PDF calling list & calling slips and manually draw the slips.
Digital Players: Click on the Play button above, and then click on the 🎫 button.
Digital Caller: Click on the Play button above.
Mixed Play: Choose any combination above.
For instance, caller can be either Printed or Digital.
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Step-By-Step:
Start by getting the Tay-Sachs Disease BINGO PDF by clicking on the "Print" button above.
Open the PDF and print it.
For random calling, you can print another copy of the call list, cut, fold and then pull them randomly at play time.
Cut the bingo cards at the cut lines if there are greater than 1 bingo cards per page.
Distribute one card per player. For marking, you can use markers. Crayons cost the least.
Choose one person to be the caller. If you are playing in a small group, the caller may as well play along with their own Bingo card.
The caller starts the game by randomly picking an item from the call list and calling out it to everyone.
The players check their cards to see if they have the called word. If they do, they cross off that word.
The first player to complete a horizontal, vertical, or a diagonal line of crossed items announces "Bingo!" and wins the game.
The caller checks that the items crossed off form a proper line according to the Bingo card and call list.
You can play for varied patterns or a full card blackout for an extended game.
This Tay-Sachs Disease BINGO Cards Game contains following Words or Phrases: 1 in 200 carriers, AAV vectors, Adult TSD = low HexA levels, Adult TSD = very aggressive, Aspiration pneumonia, Autosomal-dominant, Autosomal-recessive, Benign lipid accumulation, Bernard Tay, Blood-brain barrier, c.533 G > A, c.533 G > R, c.553 G > A, Catalytic dysfunction, Cherry-red spot, Chromosome 15q13.3, Chromosome 15q23, Chromosome 5q23, Enzyme reduction therapy, Flexible loop, Founder effect, Glucose metabolic disorder, GLUT1, Glycosphingolipid, GM2 association protein, GM2 ganglioside accumulation, GM2 ganglioside depletion, GM2-gangliosidosis, GM3 ganglioside accumulation, GM3-gangliosidosis, Gross deletion, HEXA = chromosome 5, HexA = lysosomal lipase enzyme, HexA synthesizes GM2 gangliosides, HexA α-subunit mutation, HexA β-subunit mutation, HEXB = chromosome 15, HexB deficiency, Homodimeric, HPβCD, Hydrolysis, Hypermyelination, Infantile onset = 4 months old, Infantile TSD = high HexA levels, iPSC cells, Isozymes, Juvenile TSD = most severe, Lysosomal storage metabolic disorder, Mechanical ventilation, Microglia, Miglustat, Missense mutations, N-acetylglucosamine, N-acetylneuraminic acid, Neuronal cells, Partial deletions, PP6D5 polymer, Protein metabolic disorder, Proteolytic processing, Rigid loop, Sex-linked, Silencing mutation, Toxic lipid accumulation, Warren Sachs, α-hexosaminidase A, αR424, β-hexosaminidase A, β-hexosaminidase B.
This Tay-Sachs Disease BINGO Cards Game contains following Words or Phrases: 1 in 200 carriers, AAV vectors, Adult TSD = low HexA levels, Adult TSD = very aggressive, Aspiration pneumonia, Autosomal-dominant, Autosomal-recessive, Benign lipid accumulation, Bernard Tay, Blood-brain barrier, c.533 G > A, c.533 G > R, c.553 G > A, Catalytic dysfunction, Cherry-red spot, Chromosome 15q13.3, Chromosome 15q23, Chromosome 5q23, Enzyme reduction therapy, Flexible loop, Founder effect, Glucose metabolic disorder, GLUT1, Glycosphingolipid, GM2 association protein, GM2 ganglioside accumulation, GM2 ganglioside depletion, GM2-gangliosidosis, GM3 ganglioside accumulation, GM3-gangliosidosis, Gross deletion, HEXA = chromosome 5, HexA = lysosomal lipase enzyme, HexA synthesizes GM2 gangliosides, HexA α-subunit mutation, HexA β-subunit mutation, HEXB = chromosome 15, HexB deficiency, Homodimeric, HPβCD, Hydrolysis, Hypermyelination, Infantile onset = 4 months old, Infantile TSD = high HexA levels, iPSC cells, Isozymes, Juvenile TSD = most severe, Lysosomal storage metabolic disorder, Mechanical ventilation, Microglia, Miglustat, Missense mutations, N-acetylglucosamine, N-acetylneuraminic acid, Neuronal cells, Partial deletions, PP6D5 polymer, Protein metabolic disorder, Proteolytic processing, Rigid loop, Sex-linked, Silencing mutation, Toxic lipid accumulation, Warren Sachs, α-hexosaminidase A, αR424, β-hexosaminidase A, β-hexosaminidase B.